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, alcohol, benzodiazepines, opioids, tricyclic carisoprodol antidepressants) may be additive, appropriate caution should be exercised with patients who take more than one of these CNS depressants simultaneously . Since the sedative effects of SOMA and other carisoprodol CNS depressants (e .
carisoprodol Drug Dependence, Withdrawal, and Abuse In the postmarketing experience with SOMA, cases of dependence, withdrawal, and abuse have been reported with prolonged use .
Most cases of dependence, withdrawal, and abuse occurred carisoprodol in patients who have had a history of addiction or who used SOMA in combination with other drugs with abuse potential .
However, there have been post-marketing adverse event carisoprodol reports of SOMA-associated abuse when used without other drugs with abuse potential .
Withdrawal symptoms have been reported following abrupt cessation carisoprodol after prolonged use .
To reduce the chance of SOMA dependence, withdrawal, or abuse, SOMA carisoprodol should be used with caution in addiction-prone patients and in patients taking other CNS depressants including alcohol, and SOMA should not be used more than two to three weeks for the relief of acute musculoskeletal discomfort .
Although most reports of SOMA-associated abuse have been seen in patients receiving concomitant drugs with abuse potential, there have been post-marketing adverse event reports of SOMA-associated abuse when used without other drugs with abuse One of the metabolites of SOMA, and one of carisoprodol its metabolites, meprobamate (a controlled substance), may cause dependence [see Clinical Pharmacology (12 .
There have been postmarketing reports carisoprodol of seizures in patients who received SOMA .
Most of these cases have occurred carisoprodol in the setting of multiple drug overdoses (including drugs of abuse, illegal drugs, and alcohol) [see Overdosage (10)] .

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